Title: Dr. Nouf AL-Rasheed.
College of Pharmacy, Riyadh, King Saud University.
Degree: PhD in cell physiology and pharmacology.
Current position: Assistant professor.
Address: P.O. Box: 16337, Riyadh: 11464, Saudi Arabia.
Tel: 00966 1 2913728 Fax: 00966 1 2913728.
Email: email@example.com OR firstname.lastname@example.org
King Saud University, College of Pharmacy
Riyadh, Saudi Arabia
· MAJOR: Bachelor degree in Pharmaceutical science.
· GPA: Excellent with honor degree (4.69/5).
· Date of Graduation: June, 1995.
King Saud University, Department of Pharmacology, College of Pharmacy
Riyadh, Saudi Arabia
· Started studying postgraduate courses in 1996 and completed with GPA 5/5.
March, 1999 I was awarded Master degree in Pharmacology.
The title of my thesis was:
A study on the cardiovascular effects of two novel lidocaine
Oct, 2002-Jul, 2006
I was awarded the PhD degree.
University of Leicester, Department of Cell Physiology and
Pharmacology, Leicester, UK.
The title of my thesis was: Receptor-mediated catecholamine
Release from chromaffin cells: The role of protein kinase C.
· Teaching assistant (demonstrator), Department of Pharmacology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
I was engaged in teaching the theoretical part and practical courses in pharmacology in the field of autonomic nervous system and central nervous system.
· Lecturer, Department of Pharmacology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
2006 - present
· Assistant professor, Department of Pharmacology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia.
Laboratory training and research experience:
· I was trained in Dr. Willars, G.B. laboratory (Molecular Biology and Cell Signalling), Department of Cell Physiology and Pharmacology, Faculty of Medicine and Biological Sciences, University of Leicester, Leicester, UK.
· I was trained in Dr. Seward, E.P. Laboratory (Electrophysiological studies for monitoring exocytosis events), Department of Biomedical Sciences, University of Sheffield, Sheffield, UK.
I gained experience in a number of pharmacological and biochemical approaches to examine cellular signalling events and exocytosis. In particular I gained experience in: immunoblotting and immunocytochemistry; confocal microscopy; live cell imaging of single-cell signalling events using Ca2+-sensitive dyes and eGFP-tagged proteins; population-based measurements of phosphoinositide signalling; cell culture of established cell lines; preparation and culture of primary cells; handling of cDNA; and determination of catecholamine release from cell populations.
· From 25 June till 25 August ,2007 I was a part of project team working to investigate the downstream signalling of insulin receptor and C-peptide at University Of Leicester, UK.
AL-Rasheed, N.M., AL-Sayed, M.I., AL-Zuhair, H.H., AL-Obaid, A.R.M. and Fatani, A.J. Effects of two newly synthesized analogues of lidocaine. Pharmacological Research (2001) April; 43: 313-319.
AL-Rasheed, N.M., Seward, E.P. and Willars, G.B. Selective GPCR-mediated activation of PKC isoforms in cultured bovine chromaffin cells. Abstract and Poster presentation was presented in British Pharmacological Society; Winter meeting, London, 16th-18th, December 2003.
Al-Rasheed, N.M., Seward, E.P. and Willars, G.B. Nicotine-mediated catecholamine release from bovine chromaffin cells: the role of Protein Kinase C. Abstract and Poster presentation was presented in the 4th focused meeting on cell signalling in British Pharmacological Society, Leicester, April 11th-12th, 2005.
PHL 351 (Pharmacology-1-): I have been taught the Pharmacological principles and sympathetic nervous system.
PHL 451 (Pharmacology-2-): Include the antipsychotic drugs, antidepressants and antimanics, NSAIDs, Opioids and antitussives.
PHL 461 (Clinical pharmacology): I have been taught the general principles of clinical pharmacology, clinical pharmacology of diabetes mellitus and transplant rejection.
PHL 511 (postgraduate course): Include measurements of arterial blood pressure and HR. Effect of agonist and antagonist. The influence of cardiac stimulants and depressants on isolated heart, and the influence of antiarrhythmic compounds on arrhythmia induced experimentally in guinea pigs.